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Creatine

  • Vitamins & Supplements
  • Updated April 9, 2024

Creatine is a derivative of the amino acids glycine, arginine, and methionine that is made in the body and obtained through food. Creatine is used as part of a reserve energy system which helps cells meet their real-time energy needs, and stave off fatigue when working at maximum capacity. Muscles have the largest creatine stores. Creatine supplementation is used in combination with strength training to boost muscle gains and is generally safe. Creatine stores in the brain may allow for more flexible energy utilization. However, the ability of creatine supplementation to influence cognition appears limited. 

Evidence

Observational studies suggest that adequate dietary intake of creatine promotes cognitive performance, while clinical studies suggest that the potential impact of additional creatine supplementation depends on baseline brain metabolic status and cognitive task complexity. Trials to date have not shown a protective effect for neurodegenerative disease. Our search identified:

  • 2 meta-analyses or systematic reviews of clinical trials assessing cognition in healthy adults
  • 3 observational studies for dietary creatine and cognition 
  • 3 clinical trials in Parkinson’s disease
  • 3 clinical trials in Huntington’s disease
  • 2 clinical trials in amyotrophic lateral sclerosis 
  • 5 neuroimaging studies for brain creatine/phosphocreatine levels

Potential Benefit

Higher dietary intake of creatine, 1 gram or more per day, has been associated with better performance on cognitive tests in people over age 60 [1; 2]. This level of intake is in line with the level needed to maintain creatine stores in muscle. However, the level of creatine in the brain is tightly regulated, such that brain creatine stores are less likely to change in response to fluctuations in dietary levels compared to muscle stores [3; 4]. For example, vegetarians tend to have lower intramuscular levels, but normal brain creatine levels [4]. Since the loss of muscle tissue in late life is itself a risk factor for dementia [5], observed associations between creatine levels and cognition could be a byproduct of better preservation of muscle, though this has not been tested directly. High dose creatine loading has been used as a reliable way of boosting up muscle creatine stores [6]. The impact of similar creatine loading paradigms on brain stores has been less consistent, only modestly boosting brain levels in those with low baseline creatine [3]. Consistent with this, the impact of creatine supplementation on cognition has been mixed in clinical trials [7]. Creatine has been more likely to show benefit when baseline levels are lower and energy demand is increased, such as in older adults, following sleep deprivation, and when performing complex cognitive tasks at a high rate of speed [8; 9]. Similar to how it is used in muscle, higher levels of creatine may delay the onset of fatigue in brain cells. 

For Dementia Patients

Due to its potential role in promoting more efficient energy utilization in the brain, creatine was expected to help maintain cognitive function in patients with neurodegenerative disease [10]. However, no clear benefits have been observed in clinical trials conducted to date in patients with Parkinson’s disease, Huntington’s disease, or amyotrophic lateral sclerosis (ALS) [11; 12; 13]. A study testing creatine supplementation in Alzheimer’s disease patients is ongoing [10]. 

Safety

Creatine has shown good safety in hundreds of clinical studies, the vast majority of which tested creatine for muscle-related outcomes. The most reported side effect is a small weight gain, primarily in men, during a high dose loading phase that is related to increased water retention in the muscle [14]. Gastrointestinal effects such as nausea or bloating have been reported, but incidence rates in clinical studies are similar to control groups [15]. Rare case reports of liver or kidney injury in young men appear to be related to the use of creatine in conjunction with cocktails of multiple other supplements and/or at extremely high doses [16]. Observational studies suggest that consumption of ≥ 2 grams of creatine per day is not associated with elevated risk for liver or kidney problems [16; 17]. However, caution is still warranted in individuals with reduced kidney function. 

NOTE: This is not a comprehensive safety evaluation or complete list of potentially harmful drug interactions. It is important to discuss safety issues with your physician before taking any new supplement or medication.

How to Use

Creatine can be obtained through food, with typical consumption around 1-2 grams per day. Enriched dietary sources include red meat, poultry, and seafood. There is currently no clinically established dose of creatine for optimizing brain stores or use in neurological disorders. The most widely used and tested form of creatine supplement for muscles is creatine monohydrate, typically taken in the form of an oral powder [14]. Traditional doses for creatine monohydrate to build muscle creatine stores include a loading phase of 20 grams per day for 5-7 days followed by a maintenance phase of 5 grams per day. Studies suggest that brain creatine stores are less responsive to supplementation and may require higher doses (i.e. 20 grams per day), but this has not been clinically verified. Excessive use of creatine supplements may decrease the body’s endogenous production of creatine, such that supplementation becomes less effective over time [8]. 

Learn More

Full scientific report (PDF) on Cognitive Vitality Reports 

For more information on the history, use, and safety see Drugs.com 

References

  1. Ostojic SM, Korovljev D, Stajer V (2021) Dietary creatine and cognitive function in U.S. adults aged 60 years and over. Aging clinical and experimental research 33, 3269-3274.
  2. Oliveira EF, Forbes SC, Borges EQ et al. (2023) Association between dietary creatine and visuospatial short-term memory in older adults. Nutrition and health 29, 731-736.
  3. Pan JW, Takahashi K (2007) Cerebral energetic effects of creatine supplementation in humans. American journal of physiology Regulatory, integrative and comparative physiology 292, R1745-1750.
  4. Solis MY, Artioli GG, Otaduy MCG et al. (2017) Effect of age, diet, and tissue type on PCr response to creatine supplementation. Journal of applied physiology (Bethesda, Md : 1985) 123, 407-414.
  5. Spauwen PJ, Murphy RA, Jónsson PV et al. (2017) Associations of fat and muscle tissue with cognitive status in older adults: the AGES-Reykjavik Study. Age and ageing 46, 250-257.
  6. Kreider RB, Kalman DS, Antonio J et al. (2017) International Society of Sports Nutrition position stand: safety and efficacy of creatine supplementation in exercise, sport, and medicine. Journal of the International Society of Sports Nutrition 14, 18.
  7. Avgerinos KI, Spyrou N, Bougioukas KI et al. (2018) Effects of creatine supplementation on cognitive function of healthy individuals: A systematic review of randomized controlled trials. Experimental gerontology 108, 166-173.
  8. Candow DG, Forbes SC, Ostojic SM et al. (2023) "Heads Up" for Creatine Supplementation and its Potential Applications for Brain Health and Function. Sports medicine (Auckland, NZ) 53, 49-65.
  9. Prokopidis K, Giannos P, Triantafyllidis KK et al. (2023) Effects of creatine supplementation on memory in healthy individuals: a systematic review and meta-analysis of randomized controlled trials. Nutrition reviews 81, 416-427.
  10. Smith AN, Morris JK, Carbuhn AF et al. (2023) Creatine as a Therapeutic Target in Alzheimer's Disease. Current developments in nutrition 7, 102011.
  11. Kieburtz K, Tilley BC, Elm JJ et al. (2015) Effect of creatine monohydrate on clinical progression in patients with Parkinson disease: a randomized clinical trial. Jama 313, 584-593.
  12. Hersch SM, Schifitto G, Oakes D et al. (2017) The CREST-E study of creatine for Huntington disease: A randomized controlled trial. Neurology 89, 594-601.
  13. Shefner JM, Cudkowicz ME, Schoenfeld D et al. (2004) A clinical trial of creatine in ALS. Neurology 63, 1656-1661.
  14. Kreider RB, Jäger R, Purpura M (2022) Bioavailability, Efficacy, Safety, and Regulatory Status of Creatine and Related Compounds: A Critical Review. Nutrients 14.
  15. de Guingand DL, Palmer KR, Snow RJ et al. (2020) Risk of Adverse Outcomes in Females Taking Oral Creatine Monohydrate: A Systematic Review and Meta-Analysis. Nutrients 12.
  16. Todorovic N, Korovljev D, Stajer V et al. (2023) Creatine consumption and liver disease manifestations in individuals aged 12 years and over. Food science & nutrition 11, 1134-1141.
  17. Ostojic SM (2021) Dietary creatine and kidney function in adult population: NHANES 2017-2018. Food science & nutrition 9, 2257-2259.